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"REC"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
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  • ¿µ¹®
    ÇѱÛ
  • rectal aganglionosis
    Á÷À幫½Å°æÀýÁõ, °ðâÀڽŰæÀý¾øÀ½Áõ
  • rectal ampulla
    Á÷À寨´ë, °ðâÀÚÆØ´ë
  • rectal anesthesia
    Á÷À帶Ãë, °ðâÀÚ¸¶Ãë
  • rectal application
    Á÷Àå³»Åõ¿©, °ðâÀÚ³»Åõ¿©
  • rectal artery
    °ðâÀÚµ¿¸Æ, Á÷À嵿¸Æ
  • rectal atresia
    Á÷ÀåÆó¼â(Áõ), °ðâÀÚÆó¼â(Áõ)
  • rectal bleeding
    Á÷ÀåÃâÇ÷, °ðâÀÚÃâÇ÷
  • rectal fistula
    Á÷Àå·ç, °ðâÀÚ»û±æ
  • rectal hernia
    Á÷ÀåÅ»Àå, °ðâÀÚÅ»Àå
  • rectal incontinence
    ´ëº¯»õ±â, ´ëº¯½Ç±Ý
  • rectal infusion
    Á÷ÀåÁÖÀÔ, °ðâÀÚÁÖÀÔ
  • rectal instillation
    Á÷ÀåÁ¡ÀûÁÖÀÔ, °ðâÀÚ¹æ¿ïÁÖÀÔ
  • rectal palpation
    Á÷Àå³»ÃËÁø, °ðâÀÚÃËÁø
  • rectal prolapse
    Á÷ÀåÅ»Ãâ(Áõ), °ðâÀÚÅ»Ãâ(Áõ)
  • rectal reflex
    Á÷Àå¹Ý»ç, °ðâÀڹݻç
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 15 ÆäÀÌÁö: 8
  • ¿µ¹®
    ÇѱÛ
  • recording kata thermometer
    ±â·Ï½ÄīŸ¿Âµµ°è
  • recovery
    ȸº¹
  • recovery quotient
    ȸº¹Áö¼ö
  • recovery room
    ȸº¹½Ç
  • recovery time
    ȸº¹½Ã°£
  • recrement
    Àç±Í¾×
  • recrudescence
    Àç¹ß
  • recrudescent typhus
    Àç¹ß¹ßÁøÆ¼Çª½º
  • recruiting response
    µ¿¿ø¹ÝÀÀ
  • recruitment
    µ¿¿ø, Á¡Áõ, ¸ðÁý, º¸Ãæ
  • recruitment factor
    µ¿¿øÀÎÀÚ
  • recruitment interval
    Á¡Áõ°£°Ý
  • recruitment pattern
    Á¡Áõ¾ç»ó
  • recruitment phenomenon
    Á¡ÁõÇö»ó, ´©°¡Çö»ó, º¸ÃæÇö»ó
  • recrystallization
    Àç°áÁ¤
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 15 ÆäÀÌÁö: 8
  • ¿µ¹®
    ÇѱÛ
  • reciprocal conduction
    »ó¹ÝÀüµµ.
  • reciprocal crossing
    »ó¹Ý±³¹è(ßÓÚãÎßÛÕ), ¿ª±³Àâ(æ½ÎßíÚ).
  • reciprocal difference
    ¿ªÂ÷ºÐ(æ½ó¬ÝÂ).
  • reciprocal hybrid
    »ó¹ÝÀâÁ¾(ßÓÚãíÚðú).
  • reciprocal inhibition
    »ó¹Ý¾ïÁ¦(ßÓÚãåäð¤).
  • reciprocal inhibition
    »óÈ£¾ïÁ¦
  • reciprocal inhibition psychotherapy
    »óÈ£Á¦ÁöÁ¤½ÅÄ¡·á.
  • reciprocal innervation
    »ó¹Ý½Å°æÁö¹è(ßÓÚããêÌèò¨ÛÕ).
  • reciprocal innervation
    »ó¹ÝÀû½Å°æÁö¹è
  • reciprocal proportion
    ¹Ýºñ·Ê(ËÑËÓËç).
  • reciprocal retention
    »ó¹ÝÁ¤Ã¼(ßÓÚãïÎôò).
  • reciprocal rhythm
    Àç±Í¸®µë
  • reciprocal shaker
    ¿Õº¹ÁøÅÁ±â(èÙÜÖòè.JÐï).
  • reciprocal shaker
    ¿Õº¹ÁøÅÁ±â
  • reciprocal system
    »ó¹Ý°è(ßÓÚãͧ).
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 15 ÆäÀÌÁö: 8
  • ¿µ¹®
    ÇѱÛ
  • recessus ileocecalis superior ³ª
    À§È¸Àå¸ÍÀå¿À¸ñ, »óȸ¸ÍÇÔ¿ä(ß¾üÞØîùèèê).
  • recessus inferior omentalis ³ª
    ¸Á³¶¾Æ·¡¿À¸ñ, ÇÏÇÔ¿ä(ù»ùèèê).
  • recessus infundibuli ³ª
    ±ò¶§±â¿À¸ñ, ´©µÎÇÔ¿ä(רÔéùèèê).
  • recessus interdentalis ³ª
    Ä¡¾Æ»çÀÌ¿À¸ñ<ÀÌ»çÀÌ¿À¸ñ>, Ä¡±Ù¿Í(öÍÐÆèÀ).
  • recessus intersigmoideus ³ª
    SÀÚ°áÀå»çÀÌ¿À¸ñ, S»ó°áÀå°£ÇÔ¿ä(¡­ßÒÌ¿ Êàùèèê ).
  • recessus isthmicus ³ª
    Á¼Àº¿À¸ñ, ÇùÇÔ¿ä( ùèèê).
  • recessus lateralis ventriculi quarti ³ª
    ³Ý°³ú½Ç¿ÜÃø¿À¸ñ, Á¦4³ú½Ç¿ÜÃøÇÔ ¿ä(ð¯ÞÌÒàãøèâö° ùèèê).
  • recessus lienalis ³ª
    ºñÀå¿À¸ñ, ºñÇÔ¿ä (Þ¡ùèèê).
  • recessus membranae tympani aboralis ³ª
    °í¸·(ÍÕØ¯).
  • recessus membranae tympani anterior ³ª
    ¾Õ°í¸·¿À¸ñ, Àü°í¸·ÇÔ¿ä(îñÍÕØ¯ùèèê ).
  • recessus membranae tympani posterior ³ª
    µÚ°í¸·¿À¸ñ, ÈÄ°í¸·ÇÔ¿ä(ý­ÍÕØ¯ùè èê).
  • recessus membranae tympani superior ³ª
    À§°í¸·¿À¸ñ, »ó°í¸·ÇÔ¿ä(ß¾ÍÕØ¯ùèèê ).
  • recessus nasopalatinus ³ª
    ÄÚ±¸°³¿Í, ºñ±¸°³ÇÔ¿ä(ެϢËÏùèèê).
  • recessus opticus ³ª
    ½Ã°¢±³Â÷¿À¸ñ, ½Ã±³Â÷ÇÔ¿ä(ãÊÎßó¬ùèèê).
  • recessus orbito ethmoidalis ³ª
    ¾È¿Í»ç°ñ¿À¸ñ, ¾È¿Í»ç°ñ¿Í(äÑèÀÞèÍéèÀ).
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  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • recurrent stomatitis
    Àç¹ß¼º ±¸³»¿°
    Àç¹ß¼º ±¸³»¿°À̶õ Á¤È®ÇÑ º´¸íÀº ¾Æ´Ï¸ç ÁÖ±âÀûÀ¸·Î ÀÕ¸öÀÌ º×°í, Çú¹Ù´ÃÀÌ µ¸°í, ¸ñÀÌ ¾ÆÇ Áõ»óÀ» À¯¹ßÇÏ´Â ¸ðµç ÁúȯÀ» ÅëĪÇÑ´Ù. º» ÁúȯÀÇ ¿øÀÎÀº ´Ù¾çÇÏ¸ç ¾ÆÁ÷ ¹àÇôÁöÁö ¾ÊÀº ºÎºÐÀÌ ¸¹´Ù. ±× Áß¿¡ ´ëÇ¥ÀûÀÎ °ÍÀº ¾ÆÇÁŸ¼º ±¸³»¿°ÀÌ ÀÖ´Ù. º» ÁúȯÀº óÀ½¿¡ ±¸°­ Á¡¸·¿¡ ¹ÝÁ¡ÀÌ »ý±â¸é¼­ ¿©±â¿¡ È«¹Ý¼º
  • recurrent tumor
    Àç¹ß¼º Á¾¾ç
  • recurring blister
    Àç¹ß¼º ¼öÆ÷
  • recurring oral ulcer
    Àç¹ß¼º ±¸°­ ±Ë¾ç
  • recurring type
    Àç¹ß¼º
CancerWEB ¿µ¿µ ÀÇÇлçÀü ¸ÂÃã °Ë»ö °á°ú : 15 ÆäÀÌÁö: 8
receptors, gaba-b Cell surface proteins which bind gaba and influence cells via interactions with g-proteins. Gaba-b receptors are pharmacologically characterised by their insensitivity to the blocker bicuculline and sensitivity to the agonist l-baclofen. They are found both presynaptically and postsynaptically, and act variously by inhibition of adenylate cyclase, activation of phospholipase a2, activation of potassium channels, and inactivation of voltage-activated calcium channels.
(12 Dec 1998)
receptors, gastrointestinal hormone Cell surface proteins that bind gastrointestinal hormones with high affinity and trigger intracellular changes influencing the behaviour of cells. most gastrointestinal hormones also act as neurotransmitters so these receptors are also present in the central and peripheral nervous systems.
(12 Dec 1998)
receptors, glucagon Cell surface receptors that bind glucagon with high affinity and trigger intracellular changes which influence the behaviour of cells. Activation of glucagon receptors causes a variety of effects; the best understood is the initiation of a complex enzymatic cascade in the liver which ultimately increases the availability of glucose to body organs.
(12 Dec 1998)
receptors, glucocorticoid Cytoplasmic proteins that specifically bind glucocorticoids and mediate their cellular effects. The glucocorticoid receptor-glucocorticoid complex acts in the nucleus to induce transcription of DNA. Glucocorticoids were named for their actions on blood glucose concentration, but they have equally important effects on protein and fat metabolism. Cortisol is the most important example.
(12 Dec 1998)
receptors, glutamate Cell-surface proteins that bind glutamate and trigger changes which influence the behaviour of cells. Glutamate receptors include ionotropic receptors (ampa, kainate, and n-methyl-d-aspartate receptors), which directly control ion channels, and metabotropic receptors which act through second messenger systems. Glutamate receptors are the most common mediators of fast excitatory synaptic transmission in the central nervous system. They have also been implicated in the mechanisms of memory and of many diseases.
(12 Dec 1998)
receptors, glycine Cell surface receptors that bind glycine with high affinity and trigger intracellular changes which influence the behaviour of cells. Glycine receptors in the central nervous system have an intrinsic chloride channel and are usually inhibitory.
(12 Dec 1998)
receptors, gonadotropin Those protein complexes or molecular sites on the surfaces of gonadal and other sensitive cells that bind gonadotropins and thereby modify the functions of those cells; hcg, lh, and fsh are the major specific gonadotropins.
(12 Dec 1998)
receptors, granulocyte-colony-stimulating factor Receptors that bind and internalise granulocyte-colony-stimulating factor. Their mw is believed to be 150 kD. These receptors are found mainly on a subset of myelomonocytic cells.
(12 Dec 1998)
receptors, granulocyte-macrophage colony-stimulating factor Receptors that bind and internalise the granulocyte-macrophage stimulating factor. Their mw is believed to be 84 kD. The most mature myelomonocytic cells, specifically human neutrophils, macrophages, and eosinophils, express the highest number of affinity receptors for this growth factor.
(12 Dec 1998)
receptors, growth factor Cell surface receptors that bind growth or trophic factors with high affinity, triggering intracellular responses which influence the growth, differentiation, or survival of cells.
(12 Dec 1998)
receptors, histamine Cell-surface proteins that bind histamine and trigger intracellular changes influencing the behaviour of cells. Histamine receptors are widespread in the central nervous system and in peripheral tissues. Three types have been recognised and designated h1, h2, and h3. They differ in pharmacology, distribution, and mode of action.
(12 Dec 1998)
receptors, histamine h1 A class of histamine receptors discriminated by their pharmacology and mode of action. most histamine h1 receptors operate through the inositol phosphate/diacylglycerol second messenger system. Among the many responses mediated by these receptors are smooth muscle contraction, increased vascular permeability, hormone release, and cerebral glyconeogenesis.
(12 Dec 1998)
receptors, histamine h2 A class of histamine receptors discriminated by their pharmacology and mode of action. Histamine h2 receptors act via g-proteins to stimulate adenylate cylase. Among the many responses mediated by these receptors are gastric acid secretion, smooth muscle relaxation, inotropic and chronotropic effects on heart muscle, and inhibition of lymphocyte function.
(12 Dec 1998)
receptors, histamine h3 A class of histamine receptors discriminated by their pharmacology and mode of action. Histamine h3 receptors were first recognised as inhibitory autoreceptors on histamine-containing nerve terminals and have since been shown to regulate the release of several neurotransmitters in the central and peripheral nervous systems.
(12 Dec 1998)
receptors, HIV Cellular receptors that bind the human immunodeficiency virus that causes aids. Included are CD4 antigens, found on t4 lymphocytes, and monocytes/macrophages, which bind to the HIV envelope protein gp120.
(12 Dec 1998)
MeSH(Medical Subject Headings) ¸ÂÃã °Ë»ö (http://www.nlm.nih.gov) °á°ú : 5 ÆäÀÌÁö: 8
  • Receptor, Epidermal Growth Factor - »õâ A cell surface receptor involved in regulation of cell growth and differentiation. It is specific for EPIDERMAL GROWTH FACTOR and EGF related peptides including TRANSFORMING GROWTH FACTOR ALPHA, amphiregulin, and heparin-binding EGF-like growth factor. The binding of ligand to the receptor causes activation of its intrinsic tyrosine kinase activity and rapid internalization of the receptor-ligand complex into the cell.
    Synonyms : Epidermal Growth Factor Receptor Kinase, Receptors, Epidermal Growth Factor, Epidermal Growth Factor Receptor Protein Tyrosine Kinase, Proto-Oncogene Protein, erbB-1, Receptor, TGF alpha, Receptor, Transforming Growth Factor alpha, c erbB 1 Protein
  • Receptor, erbB-2 - »õâ A cell surface protein-tyrosine kinase receptor that is found to be overexpressed in a significant number of adenocarcinomas. It has extensive homology to and can heterodimerize with the EGF receptor (RECEPTOR, EPIDERMAL GROWTH FACTOR), the erbB-3 receptor (RECEPTOR, ERBB-3) and the erbB-4 receptor. Activation of the erbB-2 receptor occurs during heterodimer formation with a ligand-bound erbB receptor family members.
    Synonyms : Oncogene Protein HER-2, Proto-Oncogene Proteins c-erbB-2, Receptors, erbB-2, p185(c-neu), HER 2 Proto Oncogene Protein, Oncogene Protein HER 2, Proto Oncogene Protein HER 2, Proto Oncogene Proteins c erbB 2, Proto-Oncogene Protein, HER-2, Receptor, erbB 2
  • Receptor, erbB-3 - »õâ A cell surface protein-tyrosine kinase receptor that is specific for NEUREGULINS. It has extensive homology to and can heterodimerize with the EGF Receptor (RECEPTOR, EPIDERMAL GROWTH FACTOR) and the erbB-2 receptor (RECEPTOR, ERBB-2). Overexpression of the erbB-3 receptor is associated with tumorigenesis.
    Synonyms : Proto-Oncogene Protein c-erbB-3, Proto-Oncogene Proteins erbB-3, HER 3 Proto Oncogene Protein, Proto Oncogene Protein c erbB 3, Proto Oncogene Protein erbB 3, Proto Oncogene Proteins erbB 3, Proto-Oncogene Protein, HER-3, Receptor, erbB 3, c erbB 3 Protein
  • Receptor, Fibroblast Growth Factor, Type 1 - »õâ A fibroblast growth factor receptor with specificity for FIBROBLAST GROWTH FACTORS; HEPARAN SULFATE PROTEOGLYCAN; and NEURONAL CELL ADHESION MOLECULES. Several variants of the receptor exist due to multiple ALTERNATIVE SPLICING of its mRNA. Fibroblast growth factor receptor 1 is a tyrosine kinase that transmits signals through the MAP KINASE SIGNALING SYSTEM.
    Synonyms : FGFR1 Protein, FGFR1 Tyrosine Kinase, Fibroblast Growth Factor Soluble Receptor 1, fms-Like Tyrosine Kinase-2, fms Like Tyrosine Kinase 2
  • Receptor, Fibroblast Growth Factor, Type 2 - »õâ A fibroblast growth factor receptor that is found in two isoforms. One receptor isoform is found in the MESENCHYME and is activated by FIBROBLAST GROWTH FACTOR 2. A second isoform of fibroblast growth factor receptor 2 is found mainly in EPITHELIAL CELLS and is activated by FIBROBLAST GROWTH FACTOR 7 and FIBROBLAST GROWTH FACTOR 10. Mutation of the gene for fibroblast growth factor receptor 2 can result in APERT SYNDROME.
    Synonyms : BEK Fibroblast Growth Factor Receptor, BEK Fibroblast Growth Factor Receptor Kinase, BEK Protein Tyrosine Kinase, Bek Fgf Receptor Kinase, Bek-Related Fibroblast Growth Factor-Receptor-1, FGFR2 Protein, FGFR2b, FGFR2c, Fibroblast Growth Factor Receptor 2b
¿ÜºÎ ¸µÅ© - Merriam-Webster's ÀÇÇлçÀü ¸ÂÃã °Ë»ö (https://www.merriam-webster.com) °á°ú: 5 ÆäÀÌÁö: 8
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rectoanal inhibitory reflex relaxation of the internal anal sphincter in response to increased pressure in the rectum; it can be tested by inflating a balloon in the lumen. It is absent in cases of congenital megacolon.
Ãâó: www.mercksource.com/pp/us/cns/cns_hl_dorlands.jspz...
recurrent brief depressive disorder short repeated episodes of depressive symptoms severe enough to qualify as major depressive episodes but of lesser duration, recurring at least once a month and not associated with the menstrual cycle.
Ãâó: www.mercksource.com/pp/us/cns/cns_hl_dorlands.jspz...
recovered memory A false memory is a memory of an event that did not happen or is a distortion of an event that did occur as determined by externally corroborated facts. If a person remembers an event that lacks another witness or corroborative physical evidence, the validity of the memory may be questioned—but not dismissed. It might be said that absence of evidence is not evidence of absence, but validation has the highest priority. ...
Ãâó: en.wikipedia.org/wiki/Recovered_memory
recall bias Taken generally, recall bias occurs when the way a survey respondent answers a question is affected not just by the correct answer, but also by the respondent's memory. This can affect the results of the survey. As a hypothetical example, suppose that a survey in 2005 asked respondents whether they believed that O. J. Simpson had killed his wife. ...
Ãâó: en.wikipedia.org/wiki/Recall_bias
recapitulation Sonata form refers to both the standard layout of an entire musical composition and more specifically to the standardized form of the first movement. The latter is also referred to as sonata-allegro form. Sonata form is both a way of organizing the composing of a work and a way of analyzing an existing work. ...
Ãâó: en.wikipedia.org/wiki/Recapitulation
ÇÑ¿µ/¿µÇÑ »çÀü ¸ÂÃã °Ë»ö °á°ú : 15 ÆäÀÌÁö: 8
  • ¿µ¹®
    ÇѱÛ
  • recession
    Åð°Å;ÈÄÅð;¾¦ µé¾î°£ °÷;¿ì¹¬ÇÑ °÷;ÈĹÌÁø °÷;(ÀϽÃÀû)°æ±â ÈÄÅð;ºÒ°æ±â
  • recessional
    ¹°·¯³¯ ¶§ÀÇ
  • recessional
    Åð°ÅÀÇ;Åð°ÅÇÒ ¶§¿¡ ºÎ¸£´Â;(ÀÇȸÀÇ)ÈÞȸÀÇ;ÈÞÁ¤ÀÇ;ÈÞ°¡ÀÇ;=RECESSIONAL HYMN;(¿¹¹è ÈÄÀÇ)ÅðÀå(ÀÇ ¿­)
  • recessional hymn
    ÅðÀå ¼º°¡(¿¹¹è ÈÄ ¸ñ»ç¿Í ÇÕâ´ë°¡ ÅðÀåÇÏ´Â µ¿¾È ºÎ¸£´Â)
  • recessionary
    °æ±â ÈÄÅðÀÇ;ºÒȲ°ú °ü·ÃµÈ
  • recessive
    ¿ªÇàÇÏ´Â; ¼èÅðÇÏ´Â
  • recessive
    ÅðÇàÀÇ;¿ªÇàÇÏ´Â;¿­¼ºÀÇ;¿­¼º ÇüÁú;~ly;~ness
  • recessive accent
    ¿ªÇà ¾Ç¼¾Æ®
  • recessive gene
    ¿­¼º À¯ÀüÀÚ
  • Rechabite
    ±ÝÁÖÀÚ(ȸ¿ø)
  • recharge
    Àç½À°Ý;¿ª½À;ÀçÀåź;ÀçÃæÀü;Àç½À°ÝÇÏ´Ù;Àç°í¹ßÇÏ´Ù;ÀçÀåÀü(ÀçÃæÀü)ÇÏ´Ù;~able
  • recharter
    Àç°è¾à;(ÁöÁ¡ µîÀÇ)½Å±Ô(¼³¸³)Àΰ¡;(ÁöÁ¡µîÀÇ)½Å±Ô¼³¸³À» Àΰ¡ÇÏ´Ù
  • recheck
    Àç°ËÅäÇÏ´Ù;Àç´ëÁ¶ÇÏ´Ù
  • recherche
    Á¤¼±µÈ; Ãß±¸ÇÏ´Â; Èñ±ÍÇÑ
  • recherche
    °ñ¶ó »ÌÀº;»©¾î³­;(¿ä¸®.Ç¥ÇöÀÌ)º°³­;¸ÚÀÖ´Â;°øµéÀÎ
WordNet ÀÏ¹Ý ¿µ¿µ »çÀü °Ë»ö °á°ú : 12 ÆäÀÌÁö: 8
REC the property of having happened or appeared not long ago
REC a time immediately before the present
REC near to or not long before the present
REC being new in a time not long past
REC of the immediate past or just previous to the present time
REC approximately the last 10,000 years
REC in the recent past
REC very recently
REC the property of having happened or appeared not long ago
REC a time immediately before the present
REC a container that is used to put or keep things in
REC an electrical (or electronic) fitting that is connected to a source of power and equipped to receive an insert
¿ÜºÎ ¸µÅ© - American Heritage Dictionary ¿µ¿µ»çÀü ¸ÂÃã °Ë»ö (https://www.ahdictionary.com) °á°ú: 5 ÆäÀÌÁö: 8
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